カリフォルニア州サンディエゴにありますSanford-Burnham Medical Research Institute (SBMRI), Chanda Labにてポスドク研究員を募集しています。RNAiライブラリー、cDNAライブラリー、バイオインフォマティクス解析を駆使してウィルス感染（HIV, influenza virus）とそれに対する宿主細胞の応答（自然免疫シグナル経路）の解析を行っています。詳細は下記とWeb Page (http://www.chandalab.org)を参照してください。自然免疫研究、ウィルス研究のバックグラウンドのある方、スクリーニング技術に興味のある方を探しています。興味のある方、ご不明な点などは井上(ainoue@sanfordburnham.org)までお気軽にお問い合わせください。

Post‐Doctoral Fellow in the Infectious & Inflammatory Disease Center, Sanford‐Burnham Institute for Medical Research, La Jolla, CA, Immediate Opening(s) in the laboratory of Dr. Sumit K. Chanda for scientists with a background in one or more of the following areas:
Molecular Virology (HIV, Influenza & other RNA Viruses) & Innate Immunology/Pattern Recognition Receptor Responses.

Projects will involve employing systems‐based technologies to establish a global understanding of host‐pathogen interactions that regulate cellular responses to viralinfection, and inhibit viral replication. Studies will integrate both large‐scale technologies (RNAi, AP‐MS, NGen seq, etc.), and more traditional cell & molecular biology and biochemistry‐based approaches. A background in systems‐ or computational biology is not essential. Positions are supported by NIH funding.

Requirements: We are seeking highly motivated and collaborative applicants with an MD, PhD, or equivalent doctoral degree, and 0‐2 years of post‐doctoral training. Prospective applications should have a made significant contributions to their area of study as evidenced by a strong publication record in high impact journals. Please visit http://www.chandalab.org for more information.

Relevant publications:
König R. et al., Human host factors required for influenza virus replication. Nature. 2010 Feb 11;463(7282):813‐7.
Bushman FD. et al., Host cell factors in HIV replication: meta‐analysis of genome‐wide studies. PLoS Pathog. 2009 May;5(5):e1000437.
König R. et al., Global analysis of host‐pathogen interactions that regulate early‐stage HIV‐1 replication. Cell. 2008 Oct 3;135(1):49‐60.
Teo H., et al. Telomere‐independent Rap1 is an IKK adaptor and regulates NF‐kB‐dependent gene expression. Nat Cell Biol. (in press).